PR001920 (Project)

Description:We report that leukemia secreted cytokines reduce miR-142 levels in T cells, causing T cell loss and exhaustion and therefore promoting CML BC transformation. Our homemade synthetic miR-142 increased T cell antileukemic activity and significantly prolonged the survival of CML BC murine and PDX models.
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Metabolomics

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A subject from Metabolomics produced as part of the PR001920 project

File

A 19.6 GB file from Metabolomics produced from OBI:0003097 as part of the PR001920 project

File

A 2.36 MB file from Metabolomics produced from OBI:0003097 as part of the PR001920 project

File

A 1.05 MB file from Metabolomics produced from OBI:0003097 as part of the PR001920 project

File

A 1.97 MB file from Metabolomics produced from OBI:0003097 as part of the PR001920 project

File

A 477 KB file from Metabolomics produced from OBI:0003097 as part of the PR001920 project

Biosample

A biosample from Metabolomics produced as part of the PR001920 project

Biosample

A biosample from Metabolomics produced as part of the PR001920 project

Biosample

A biosample from Metabolomics produced as part of the PR001920 project


  • Subject

    A subject from Metabolomics produced as part of the PR001920 project


  • File

    A 19.6 GB file from Metabolomics produced from OBI:0003097 as part of the PR001920 project


  • File

    A 2.36 MB file from Metabolomics produced from OBI:0003097 as part of the PR001920 project


  • File

    A 1.05 MB file from Metabolomics produced from OBI:0003097 as part of the PR001920 project


  • File

    A 1.97 MB file from Metabolomics produced from OBI:0003097 as part of the PR001920 project


  • File

    A 477 KB file from Metabolomics produced from OBI:0003097 as part of the PR001920 project


  • Biosample

    A biosample from Metabolomics produced as part of the PR001920 project


  • Biosample

    A biosample from Metabolomics produced as part of the PR001920 project


  • Biosample

    A biosample from Metabolomics produced as part of the PR001920 project

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